By Roger S. Kirby MD FRCS(Urol) FEBU
It is a condensed model of the author's profitable Atlas of Prostatic Disease. The emphasis is on a succinct description of prostatic sickness in a mostly illustrative layout. The booklet covers the explanations, analysis and staging of prostate melanoma, benign prostatic hyperplasia and prostatitis. The sections on therapy were more advantageous via the addition of algorithms to summarize the decision-making technique.
Men's health and wellbeing is now an incredible public wellbeing and fitness factor and prostatic sickness could have a big impression at the caliber of lifetime of many males. This publication will supply a handy precis for urologists, fundamental care physicians and nurse practitioners
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Mutation or deletion of the gene thus encourages uncontrolled cell division suppressor genes16, the best examples of which are the p53 and retinoblastoma tumor suppressor genes17,18. The p53 protein encoded by the former gene acts as an important regulator of cell division. Point mutation or complete deletion of this gene permits abnormal cell proliferation to occur (Figure 20). The p53 tumor suppressor gene has also been implicated as an important factor in the development of other cancers, including lung, breast, colon and bladder neoplasms19,20.
Deletion of the gene encoding either of these important proteins may facilitate the metastatic process by allowing malignant cells to migrate into the lymphatics and blood-stream (Figure 21). Loss of E-cadherin-staining in prostate cancer specimens appears to be strongly correlated with the subsequent development of metastases and is associated with a poor prognosis in prostate cancer patients. Angiogenesis factors For prostate cancer metastases to develop, tumor cells not only have to be released into lymphatics or the blood circulation, but cells must also have the ability to implant elsewhere and grow.
In approximately 10% of cases, this is the result of a bacterial, chlamydial or other infecting microorganisms. For the remaining 90% of cases, however, no definite etiological cause has yet been identified. Prostatic ducts, and the acini into which they branch (see Figure 2), are the foci of these inflammatory processes. A number of non-infective etiological factors may be involved. When inflammation does occur, the subsequent chronic prostatitis affects the peripheral zone more often than the central zone of the gland.
An illustrated pocketbook of prostatic diseases by Roger S. Kirby MD FRCS(Urol) FEBU